Hernandez, F; Shull, P B; Camarillo, D B
Evaluation of a laboratory model of human head impact biomechanics Journal Article
In: Journal of Biomechanics, vol. 48, no. 12, pp. 3469–3477, 2015.
Abstract | BibTeX | Tags: *HEAD, *Laboratories, *Mechanical Phenomena, *Models, Acceleration, Biological, Biomechanical Phenomena, Brain Concussion/et [Etiology], Football/in [Injuries], Head Protective Devices, Humans, Male, Neck/ph [Physiology], Rotation, SAFETY
@article{Hernandez2015,
title = {Evaluation of a laboratory model of human head impact biomechanics},
author = {Hernandez, F and Shull, P B and Camarillo, D B},
year = {2015},
date = {2015-01-01},
journal = {Journal of Biomechanics},
volume = {48},
number = {12},
pages = {3469--3477},
abstract = {This work describes methodology for evaluating laboratory models of head impact biomechanics. Using this methodology, we investigated: how closely does twin-wire drop testing model head rotation in American football impacts? Head rotation is believed to cause mild traumatic brain injury (mTBI) but helmet safety standards only model head translations believed to cause severe TBI. It is unknown whether laboratory head impact models in safety standards, like twin-wire drop testing, reproduce six degree-of-freedom (6DOF) head impact biomechanics that may cause mTBI. We compared 6DOF measurements of 421 American football head impacts to twin-wire drop tests at impact sites and velocities weighted to represent typical field exposure. The highest rotational velocities produced by drop testing were the 74th percentile of non-injury field impacts. For a given translational acceleration level, drop testing underestimated field rotational acceleration by 46% and rotational velocity by 72%. Primary rotational acceleration frequencies were much larger in drop tests ($sim$100 Hz) than field impacts ($sim$10 Hz). Drop testing was physically unable to produce acceleration directions common in field impacts. Initial conditions of a single field impact were highly resolved in stereo high-speed video and reconstructed in a drop test. Reconstruction results reflected aggregate trends of lower amplitude rotational velocity and higher frequency rotational acceleration in drop testing, apparently due to twin-wire constraints and the absence of a neck. These results suggest twin-wire drop testing is limited in modeling head rotation during impact, and motivate continued evaluation of head impact models to ensure helmets are tested under conditions that may cause mTBI. Copyright © 2015 Elsevier Ltd. All rights reserved.},
keywords = {*HEAD, *Laboratories, *Mechanical Phenomena, *Models, Acceleration, Biological, Biomechanical Phenomena, Brain Concussion/et [Etiology], Football/in [Injuries], Head Protective Devices, Humans, Male, Neck/ph [Physiology], Rotation, SAFETY},
pubstate = {published},
tppubtype = {article}
}
Ivancic, P C
Neck injury response to direct head impact Journal Article
In: Accident Analysis & Prevention, vol. 50, pp. 323–329, 2013.
Abstract | BibTeX | Tags: *Accidents, *Neck Injuries/et [Etiology], *Neck Injuries/pp [Physiopathology], Acceleration, ANALYSIS of variance, Biomechanical Phenomena, Cadaver, Humans, Manikins, Rotation, Traffic, VIDEO recording
@article{Ivancic2013,
title = {Neck injury response to direct head impact},
author = {Ivancic, P C},
year = {2013},
date = {2013-01-01},
journal = {Accident Analysis \& Prevention},
volume = {50},
pages = {323--329},
abstract = {Previous in vivo studies have observed flexion of the upper or upper/middle cervical spine and extension at inferior spinal levels due to direct head impacts. These studies hypothesized that hyperflexion may contribute to injury of the upper or middle cervical spine during real-life head impact. Our objectives were to determine the cervical spine injury response to direct head impact, document injuries, and compare our results with previously reported in vivo data. Our model consisted of a human cadaver neck (n=6) mounted to the torso of a rear impact dummy and carrying a surrogate head. Rearward force was applied to the model's forehead using a cable and pulley system and free-falling mass of 3.6kg followed by 16.7kg. High-speed digital cameras tracked head, vertebral, and pelvic motions. Average peak spinal rotations observed during impact were statistically compared (P\<0.05) to physiological ranges obtained from intact flexibility tests. Peak head impact force was 249 and 504N for the 3.6 and 16.7kg free-falling masses, respectively. Occipital condyle loads reached 205.3N posterior shear, 331.4N compression, and 7.4Nm extension moment. We observed significant increases in intervertebral extension peaks above physiologic at C6/7 (26.3degree vs. 5.7degree) and C7/T1 (29.7degree vs. 4.6degree) and macroscopic ligamentous and osseous injuries at C6 through T1 due to the 504N impacts. Our results indicate that a rearward head shear force causes complex neck loads of posterior shear, compression, and extension moment sufficient to injure the lower cervical spine. Real-life neck injuries due to motor vehicle crashes, sports impacts, or falls are likely due to combined loads transferred to the neck by direct head impact and torso inertial loads. Copyright © 2012 Elsevier Ltd. All rights reserved.},
keywords = {*Accidents, *Neck Injuries/et [Etiology], *Neck Injuries/pp [Physiopathology], Acceleration, ANALYSIS of variance, Biomechanical Phenomena, Cadaver, Humans, Manikins, Rotation, Traffic, VIDEO recording},
pubstate = {published},
tppubtype = {article}
}
Hamberger, A; Huang, Y L; Zhu, H; Bao, F; Ding, M; Blennow, K; Olsson, A; Hansson, H A; Viano, D; Haglid, K G
Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head Journal Article
In: Journal of Neurotrauma, vol. 20, no. 2, pp. 169–178, 2003.
Abstract | BibTeX | Tags: *Amyloid beta-Peptides/me [Metabolism], *Brain Injuries/me [Metabolism], *Brain/me [Metabolism], *Neurofilament Proteins/me [Metabolism], 0 (Amyloid beta-Peptides), 0 (neurofilament protein L), 0 (Neurofilament Proteins), 108688-71-7 (neurofilament protein H), Acceleration, Animals, Brain Injuries/et [Etiology], immunohistochemistry, Phosphorylation, Rabbits, Rotation, Tissue Distribution
@article{Hamberger2003,
title = {Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head},
author = {Hamberger, A and Huang, Y L and Zhu, H and Bao, F and Ding, M and Blennow, K and Olsson, A and Hansson, H A and Viano, D and Haglid, K G},
year = {2003},
date = {2003-01-01},
journal = {Journal of Neurotrauma},
volume = {20},
number = {2},
pages = {169--178},
abstract = {Rotational acceleration of the head, as occurs in falls, car crashes, and sport injuries, may result in diffuse brain damage, with acute and chronic neurological and psychiatric symptoms. The present study addresses the effects of rotational trauma on the neuronal cytoskeleton, which stabilizes perikaryal, dendritic and axonal shape and function. The study focuses upon the distribution of (1) the phosphorylated form of the heavy neurofilament subunit, (2) the light neurofilament subunit, and (3) beta-amyloid, a marker for brain injury. While normally restricted to axons, the phosphorylated heavy neurofilament subunits were drastically decreased in the axons after rotational trauma. Instead, they accumulated in the neuronal perikarya, normally devoid of the phosphorylated subunit. This alteration was seen, not only in the cerebral cortex, but also in the hippocampus, the cervical spinal cord, the cerebellum, the cranial nerves and the pyramidal tract. The distribution of the light subunit of neurofilaments was also altered post trauma. Only a weak beta-amyloid immunoreactivity was detected in the brains of control animals. Promptly after the trauma, a large number of beta-amyloid positive neurons appeared. Intensely co-localized immunoreactivity for the light subunit of neurofilaments and of beta-amyloid was seen 3 days after the rotational trauma axons of in the subcortical white matter and in the granule cell layer of the dentate gyrus as well as in neurons of the hypoglossal nucleus. The reported alterations in the central nervous system neurons are similar to those in the human brain after closed head injury and in chronic degenerative diseases. Regions of importance for social behavior, memory and body movement were affected.},
keywords = {*Amyloid beta-Peptides/me [Metabolism], *Brain Injuries/me [Metabolism], *Brain/me [Metabolism], *Neurofilament Proteins/me [Metabolism], 0 (Amyloid beta-Peptides), 0 (neurofilament protein L), 0 (Neurofilament Proteins), 108688-71-7 (neurofilament protein H), Acceleration, Animals, Brain Injuries/et [Etiology], immunohistochemistry, Phosphorylation, Rabbits, Rotation, Tissue Distribution},
pubstate = {published},
tppubtype = {article}
}
Corzatt, R D; Groppel, J L; Pfautsch, E; Boscardin, J
The biomechanics of head-first versus feet-first sliding Journal Article
In: American Journal of Sports Medicine, vol. 12, no. 3, pp. 229–232, 1984.
Abstract | BibTeX | Tags: *Baseball, *Biomechanical Phenomena, *SPORTS, Evaluation Studies as Topic, Foot/ph [Physiology], Head/ph [Physiology], Humans, Motion Pictures as Topic, Rotation, Time Factors
@article{Corzatt1984,
title = {The biomechanics of head-first versus feet-first sliding},
author = {Corzatt, R D and Groppel, J L and Pfautsch, E and Boscardin, J},
year = {1984},
date = {1984-01-01},
journal = {American Journal of Sports Medicine},
volume = {12},
number = {3},
pages = {229--232},
abstract = {The two basic sliding techniques, head-first and feet-first, were analyzed kinematically with high speed cinematography. Four phases were identified with each technique: sprint, attainment of sliding position, airborne, and landing. The velocity and displacement of the center of gravity were measured with each technique. The study was primarily descriptive but demonstrated potential for injury with each technique. Further studies are needed to determine which technique is safer and faster.},
keywords = {*Baseball, *Biomechanical Phenomena, *SPORTS, Evaluation Studies as Topic, Foot/ph [Physiology], Head/ph [Physiology], Humans, Motion Pictures as Topic, Rotation, Time Factors},
pubstate = {published},
tppubtype = {article}
}
Hernandez, F; Shull, P B; Camarillo, D B
Evaluation of a laboratory model of human head impact biomechanics Journal Article
In: Journal of Biomechanics, vol. 48, no. 12, pp. 3469–3477, 2015.
@article{Hernandez2015,
title = {Evaluation of a laboratory model of human head impact biomechanics},
author = {Hernandez, F and Shull, P B and Camarillo, D B},
year = {2015},
date = {2015-01-01},
journal = {Journal of Biomechanics},
volume = {48},
number = {12},
pages = {3469--3477},
abstract = {This work describes methodology for evaluating laboratory models of head impact biomechanics. Using this methodology, we investigated: how closely does twin-wire drop testing model head rotation in American football impacts? Head rotation is believed to cause mild traumatic brain injury (mTBI) but helmet safety standards only model head translations believed to cause severe TBI. It is unknown whether laboratory head impact models in safety standards, like twin-wire drop testing, reproduce six degree-of-freedom (6DOF) head impact biomechanics that may cause mTBI. We compared 6DOF measurements of 421 American football head impacts to twin-wire drop tests at impact sites and velocities weighted to represent typical field exposure. The highest rotational velocities produced by drop testing were the 74th percentile of non-injury field impacts. For a given translational acceleration level, drop testing underestimated field rotational acceleration by 46% and rotational velocity by 72%. Primary rotational acceleration frequencies were much larger in drop tests ($sim$100 Hz) than field impacts ($sim$10 Hz). Drop testing was physically unable to produce acceleration directions common in field impacts. Initial conditions of a single field impact were highly resolved in stereo high-speed video and reconstructed in a drop test. Reconstruction results reflected aggregate trends of lower amplitude rotational velocity and higher frequency rotational acceleration in drop testing, apparently due to twin-wire constraints and the absence of a neck. These results suggest twin-wire drop testing is limited in modeling head rotation during impact, and motivate continued evaluation of head impact models to ensure helmets are tested under conditions that may cause mTBI. Copyright © 2015 Elsevier Ltd. All rights reserved.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Ivancic, P C
Neck injury response to direct head impact Journal Article
In: Accident Analysis & Prevention, vol. 50, pp. 323–329, 2013.
@article{Ivancic2013,
title = {Neck injury response to direct head impact},
author = {Ivancic, P C},
year = {2013},
date = {2013-01-01},
journal = {Accident Analysis \& Prevention},
volume = {50},
pages = {323--329},
abstract = {Previous in vivo studies have observed flexion of the upper or upper/middle cervical spine and extension at inferior spinal levels due to direct head impacts. These studies hypothesized that hyperflexion may contribute to injury of the upper or middle cervical spine during real-life head impact. Our objectives were to determine the cervical spine injury response to direct head impact, document injuries, and compare our results with previously reported in vivo data. Our model consisted of a human cadaver neck (n=6) mounted to the torso of a rear impact dummy and carrying a surrogate head. Rearward force was applied to the model's forehead using a cable and pulley system and free-falling mass of 3.6kg followed by 16.7kg. High-speed digital cameras tracked head, vertebral, and pelvic motions. Average peak spinal rotations observed during impact were statistically compared (P\<0.05) to physiological ranges obtained from intact flexibility tests. Peak head impact force was 249 and 504N for the 3.6 and 16.7kg free-falling masses, respectively. Occipital condyle loads reached 205.3N posterior shear, 331.4N compression, and 7.4Nm extension moment. We observed significant increases in intervertebral extension peaks above physiologic at C6/7 (26.3degree vs. 5.7degree) and C7/T1 (29.7degree vs. 4.6degree) and macroscopic ligamentous and osseous injuries at C6 through T1 due to the 504N impacts. Our results indicate that a rearward head shear force causes complex neck loads of posterior shear, compression, and extension moment sufficient to injure the lower cervical spine. Real-life neck injuries due to motor vehicle crashes, sports impacts, or falls are likely due to combined loads transferred to the neck by direct head impact and torso inertial loads. Copyright © 2012 Elsevier Ltd. All rights reserved.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Hamberger, A; Huang, Y L; Zhu, H; Bao, F; Ding, M; Blennow, K; Olsson, A; Hansson, H A; Viano, D; Haglid, K G
Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head Journal Article
In: Journal of Neurotrauma, vol. 20, no. 2, pp. 169–178, 2003.
@article{Hamberger2003,
title = {Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head},
author = {Hamberger, A and Huang, Y L and Zhu, H and Bao, F and Ding, M and Blennow, K and Olsson, A and Hansson, H A and Viano, D and Haglid, K G},
year = {2003},
date = {2003-01-01},
journal = {Journal of Neurotrauma},
volume = {20},
number = {2},
pages = {169--178},
abstract = {Rotational acceleration of the head, as occurs in falls, car crashes, and sport injuries, may result in diffuse brain damage, with acute and chronic neurological and psychiatric symptoms. The present study addresses the effects of rotational trauma on the neuronal cytoskeleton, which stabilizes perikaryal, dendritic and axonal shape and function. The study focuses upon the distribution of (1) the phosphorylated form of the heavy neurofilament subunit, (2) the light neurofilament subunit, and (3) beta-amyloid, a marker for brain injury. While normally restricted to axons, the phosphorylated heavy neurofilament subunits were drastically decreased in the axons after rotational trauma. Instead, they accumulated in the neuronal perikarya, normally devoid of the phosphorylated subunit. This alteration was seen, not only in the cerebral cortex, but also in the hippocampus, the cervical spinal cord, the cerebellum, the cranial nerves and the pyramidal tract. The distribution of the light subunit of neurofilaments was also altered post trauma. Only a weak beta-amyloid immunoreactivity was detected in the brains of control animals. Promptly after the trauma, a large number of beta-amyloid positive neurons appeared. Intensely co-localized immunoreactivity for the light subunit of neurofilaments and of beta-amyloid was seen 3 days after the rotational trauma axons of in the subcortical white matter and in the granule cell layer of the dentate gyrus as well as in neurons of the hypoglossal nucleus. The reported alterations in the central nervous system neurons are similar to those in the human brain after closed head injury and in chronic degenerative diseases. Regions of importance for social behavior, memory and body movement were affected.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Corzatt, R D; Groppel, J L; Pfautsch, E; Boscardin, J
The biomechanics of head-first versus feet-first sliding Journal Article
In: American Journal of Sports Medicine, vol. 12, no. 3, pp. 229–232, 1984.
@article{Corzatt1984,
title = {The biomechanics of head-first versus feet-first sliding},
author = {Corzatt, R D and Groppel, J L and Pfautsch, E and Boscardin, J},
year = {1984},
date = {1984-01-01},
journal = {American Journal of Sports Medicine},
volume = {12},
number = {3},
pages = {229--232},
abstract = {The two basic sliding techniques, head-first and feet-first, were analyzed kinematically with high speed cinematography. Four phases were identified with each technique: sprint, attainment of sliding position, airborne, and landing. The velocity and displacement of the center of gravity were measured with each technique. The study was primarily descriptive but demonstrated potential for injury with each technique. Further studies are needed to determine which technique is safer and faster.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Hernandez, F; Shull, P B; Camarillo, D B
Evaluation of a laboratory model of human head impact biomechanics Journal Article
In: Journal of Biomechanics, vol. 48, no. 12, pp. 3469–3477, 2015.
Abstract | BibTeX | Tags: *HEAD, *Laboratories, *Mechanical Phenomena, *Models, Acceleration, Biological, Biomechanical Phenomena, Brain Concussion/et [Etiology], Football/in [Injuries], Head Protective Devices, Humans, Male, Neck/ph [Physiology], Rotation, SAFETY
@article{Hernandez2015,
title = {Evaluation of a laboratory model of human head impact biomechanics},
author = {Hernandez, F and Shull, P B and Camarillo, D B},
year = {2015},
date = {2015-01-01},
journal = {Journal of Biomechanics},
volume = {48},
number = {12},
pages = {3469--3477},
abstract = {This work describes methodology for evaluating laboratory models of head impact biomechanics. Using this methodology, we investigated: how closely does twin-wire drop testing model head rotation in American football impacts? Head rotation is believed to cause mild traumatic brain injury (mTBI) but helmet safety standards only model head translations believed to cause severe TBI. It is unknown whether laboratory head impact models in safety standards, like twin-wire drop testing, reproduce six degree-of-freedom (6DOF) head impact biomechanics that may cause mTBI. We compared 6DOF measurements of 421 American football head impacts to twin-wire drop tests at impact sites and velocities weighted to represent typical field exposure. The highest rotational velocities produced by drop testing were the 74th percentile of non-injury field impacts. For a given translational acceleration level, drop testing underestimated field rotational acceleration by 46% and rotational velocity by 72%. Primary rotational acceleration frequencies were much larger in drop tests ($sim$100 Hz) than field impacts ($sim$10 Hz). Drop testing was physically unable to produce acceleration directions common in field impacts. Initial conditions of a single field impact were highly resolved in stereo high-speed video and reconstructed in a drop test. Reconstruction results reflected aggregate trends of lower amplitude rotational velocity and higher frequency rotational acceleration in drop testing, apparently due to twin-wire constraints and the absence of a neck. These results suggest twin-wire drop testing is limited in modeling head rotation during impact, and motivate continued evaluation of head impact models to ensure helmets are tested under conditions that may cause mTBI. Copyright © 2015 Elsevier Ltd. All rights reserved.},
keywords = {*HEAD, *Laboratories, *Mechanical Phenomena, *Models, Acceleration, Biological, Biomechanical Phenomena, Brain Concussion/et [Etiology], Football/in [Injuries], Head Protective Devices, Humans, Male, Neck/ph [Physiology], Rotation, SAFETY},
pubstate = {published},
tppubtype = {article}
}
Ivancic, P C
Neck injury response to direct head impact Journal Article
In: Accident Analysis & Prevention, vol. 50, pp. 323–329, 2013.
Abstract | BibTeX | Tags: *Accidents, *Neck Injuries/et [Etiology], *Neck Injuries/pp [Physiopathology], Acceleration, ANALYSIS of variance, Biomechanical Phenomena, Cadaver, Humans, Manikins, Rotation, Traffic, VIDEO recording
@article{Ivancic2013,
title = {Neck injury response to direct head impact},
author = {Ivancic, P C},
year = {2013},
date = {2013-01-01},
journal = {Accident Analysis \& Prevention},
volume = {50},
pages = {323--329},
abstract = {Previous in vivo studies have observed flexion of the upper or upper/middle cervical spine and extension at inferior spinal levels due to direct head impacts. These studies hypothesized that hyperflexion may contribute to injury of the upper or middle cervical spine during real-life head impact. Our objectives were to determine the cervical spine injury response to direct head impact, document injuries, and compare our results with previously reported in vivo data. Our model consisted of a human cadaver neck (n=6) mounted to the torso of a rear impact dummy and carrying a surrogate head. Rearward force was applied to the model's forehead using a cable and pulley system and free-falling mass of 3.6kg followed by 16.7kg. High-speed digital cameras tracked head, vertebral, and pelvic motions. Average peak spinal rotations observed during impact were statistically compared (P\<0.05) to physiological ranges obtained from intact flexibility tests. Peak head impact force was 249 and 504N for the 3.6 and 16.7kg free-falling masses, respectively. Occipital condyle loads reached 205.3N posterior shear, 331.4N compression, and 7.4Nm extension moment. We observed significant increases in intervertebral extension peaks above physiologic at C6/7 (26.3degree vs. 5.7degree) and C7/T1 (29.7degree vs. 4.6degree) and macroscopic ligamentous and osseous injuries at C6 through T1 due to the 504N impacts. Our results indicate that a rearward head shear force causes complex neck loads of posterior shear, compression, and extension moment sufficient to injure the lower cervical spine. Real-life neck injuries due to motor vehicle crashes, sports impacts, or falls are likely due to combined loads transferred to the neck by direct head impact and torso inertial loads. Copyright © 2012 Elsevier Ltd. All rights reserved.},
keywords = {*Accidents, *Neck Injuries/et [Etiology], *Neck Injuries/pp [Physiopathology], Acceleration, ANALYSIS of variance, Biomechanical Phenomena, Cadaver, Humans, Manikins, Rotation, Traffic, VIDEO recording},
pubstate = {published},
tppubtype = {article}
}
Hamberger, A; Huang, Y L; Zhu, H; Bao, F; Ding, M; Blennow, K; Olsson, A; Hansson, H A; Viano, D; Haglid, K G
Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head Journal Article
In: Journal of Neurotrauma, vol. 20, no. 2, pp. 169–178, 2003.
Abstract | BibTeX | Tags: *Amyloid beta-Peptides/me [Metabolism], *Brain Injuries/me [Metabolism], *Brain/me [Metabolism], *Neurofilament Proteins/me [Metabolism], 0 (Amyloid beta-Peptides), 0 (neurofilament protein L), 0 (Neurofilament Proteins), 108688-71-7 (neurofilament protein H), Acceleration, Animals, Brain Injuries/et [Etiology], immunohistochemistry, Phosphorylation, Rabbits, Rotation, Tissue Distribution
@article{Hamberger2003,
title = {Redistribution of neurofilaments and accumulation of beta-amyloid protein after brain injury by rotational acceleration of the head},
author = {Hamberger, A and Huang, Y L and Zhu, H and Bao, F and Ding, M and Blennow, K and Olsson, A and Hansson, H A and Viano, D and Haglid, K G},
year = {2003},
date = {2003-01-01},
journal = {Journal of Neurotrauma},
volume = {20},
number = {2},
pages = {169--178},
abstract = {Rotational acceleration of the head, as occurs in falls, car crashes, and sport injuries, may result in diffuse brain damage, with acute and chronic neurological and psychiatric symptoms. The present study addresses the effects of rotational trauma on the neuronal cytoskeleton, which stabilizes perikaryal, dendritic and axonal shape and function. The study focuses upon the distribution of (1) the phosphorylated form of the heavy neurofilament subunit, (2) the light neurofilament subunit, and (3) beta-amyloid, a marker for brain injury. While normally restricted to axons, the phosphorylated heavy neurofilament subunits were drastically decreased in the axons after rotational trauma. Instead, they accumulated in the neuronal perikarya, normally devoid of the phosphorylated subunit. This alteration was seen, not only in the cerebral cortex, but also in the hippocampus, the cervical spinal cord, the cerebellum, the cranial nerves and the pyramidal tract. The distribution of the light subunit of neurofilaments was also altered post trauma. Only a weak beta-amyloid immunoreactivity was detected in the brains of control animals. Promptly after the trauma, a large number of beta-amyloid positive neurons appeared. Intensely co-localized immunoreactivity for the light subunit of neurofilaments and of beta-amyloid was seen 3 days after the rotational trauma axons of in the subcortical white matter and in the granule cell layer of the dentate gyrus as well as in neurons of the hypoglossal nucleus. The reported alterations in the central nervous system neurons are similar to those in the human brain after closed head injury and in chronic degenerative diseases. Regions of importance for social behavior, memory and body movement were affected.},
keywords = {*Amyloid beta-Peptides/me [Metabolism], *Brain Injuries/me [Metabolism], *Brain/me [Metabolism], *Neurofilament Proteins/me [Metabolism], 0 (Amyloid beta-Peptides), 0 (neurofilament protein L), 0 (Neurofilament Proteins), 108688-71-7 (neurofilament protein H), Acceleration, Animals, Brain Injuries/et [Etiology], immunohistochemistry, Phosphorylation, Rabbits, Rotation, Tissue Distribution},
pubstate = {published},
tppubtype = {article}
}
Corzatt, R D; Groppel, J L; Pfautsch, E; Boscardin, J
The biomechanics of head-first versus feet-first sliding Journal Article
In: American Journal of Sports Medicine, vol. 12, no. 3, pp. 229–232, 1984.
Abstract | BibTeX | Tags: *Baseball, *Biomechanical Phenomena, *SPORTS, Evaluation Studies as Topic, Foot/ph [Physiology], Head/ph [Physiology], Humans, Motion Pictures as Topic, Rotation, Time Factors
@article{Corzatt1984,
title = {The biomechanics of head-first versus feet-first sliding},
author = {Corzatt, R D and Groppel, J L and Pfautsch, E and Boscardin, J},
year = {1984},
date = {1984-01-01},
journal = {American Journal of Sports Medicine},
volume = {12},
number = {3},
pages = {229--232},
abstract = {The two basic sliding techniques, head-first and feet-first, were analyzed kinematically with high speed cinematography. Four phases were identified with each technique: sprint, attainment of sliding position, airborne, and landing. The velocity and displacement of the center of gravity were measured with each technique. The study was primarily descriptive but demonstrated potential for injury with each technique. Further studies are needed to determine which technique is safer and faster.},
keywords = {*Baseball, *Biomechanical Phenomena, *SPORTS, Evaluation Studies as Topic, Foot/ph [Physiology], Head/ph [Physiology], Humans, Motion Pictures as Topic, Rotation, Time Factors},
pubstate = {published},
tppubtype = {article}
}