Davis, A E
Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations Journal Article
In: Critical Care Nursing Quarterly, vol. 23, no. 3, pp. 1–13, 2000.
Abstract | BibTeX | Tags: *Brain Injuries/pa [Pathology], *Brain Injuries/pp [Physiopathology], Biomechanical Phenomena, Brain Injuries/cl [Classification], Brain Injuries/co [Complications], Brain/me [Metabolism], Brain/pa [Pathology], Humans, intracranial hemorrhage, Neurons/me [Metabolism], Neurons/pa [Pathology], Nonpenetrating/pa [Pathology], Nonpenetrating/pp [Physiopathology], Skull Fractures/pa [Pathology], Skull Fractures/pp [Physiopathology], Traumatic/pa [Pathology], Traumatic/pp [Physiopatho, Wounds
@article{Davis2000,
title = {Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations},
author = {Davis, A E},
year = {2000},
date = {2000-01-01},
journal = {Critical Care Nursing Quarterly},
volume = {23},
number = {3},
pages = {1--13},
abstract = {Traumatic brain injury (TBI) is a public health problem of great concern, because it affects more than 2 million individuals each year. TBI occurs as a result of motor vehicle crashes, falls, and sports-related events. Biomechanical mechanisms occurring at the time of the injury initiate primary and secondary injuries that evolve over several days. In this article the relationship between an blunt injury event and the subsequent damage produced is addressed. Mechanisms of brain injury from biomechanics to cellular pathobiology are presented. Primary and secondary injuries are differentiated, and specific focal and diffuse clinical syndromes are described. Cellular mechanisms responsible for injury are also addressed, because they provide the unifying concepts across the many clinical syndromes so often discussed separately in reviews of traumatic brain injury. [References: 26]},
keywords = {*Brain Injuries/pa [Pathology], *Brain Injuries/pp [Physiopathology], Biomechanical Phenomena, Brain Injuries/cl [Classification], Brain Injuries/co [Complications], Brain/me [Metabolism], Brain/pa [Pathology], Humans, intracranial hemorrhage, Neurons/me [Metabolism], Neurons/pa [Pathology], Nonpenetrating/pa [Pathology], Nonpenetrating/pp [Physiopathology], Skull Fractures/pa [Pathology], Skull Fractures/pp [Physiopathology], Traumatic/pa [Pathology], Traumatic/pp [Physiopatho, Wounds},
pubstate = {published},
tppubtype = {article}
}
McLean, A J
Brain injury without head impact? Journal Article
In: Journal of Neurotrauma, vol. 12, no. 4, pp. 621–625, 1995.
Abstract | BibTeX | Tags: *Brain Injuries/et [Etiology], Acceleration, Accidents, autopsy, Brain Injuries/mo [Mortality], cause of death, Head, Humans, Mechanical, Nonpenetrating, Stress, Traffic, Wounds
@article{McLean1995,
title = {Brain injury without head impact?},
author = {McLean, A J},
year = {1995},
date = {1995-01-01},
journal = {Journal of Neurotrauma},
volume = {12},
number = {4},
pages = {621--625},
abstract = {The proposition that acceleration of the brain without direct impact to the head can result in brain injury is examined by reviewing a series of 414 road users who were fatally injured in the vicinity of Adelaide, South Australia. The series comprises 170 pedestrians, 10 pedal cyclists, 143 motorcyclists, and 91 vehicle occupants. In each case a member of the research team attended the autopsy to look for evidence of impact on the body, particularly to the head or face. The brain was examined by a neuropathologist and the type and pattern of injury was recorded. The circumstances of the crash were investigated, including an examination of the crash site and the vehicles involved and, where relevant, interviews with witnesses. In cases involving a motorcyclist the helmet worn was retrieved by the police and assigned to the research unit for examination. Particular attention was paid to the identification of objects causing injury to the head or face and also to objects impacted by a helmet. Brain injury was recorded as a cause of death in 55% of the 403 cases for which there was a clear classification of cause of death. Brain injury, at any level of severity, was identified by a neuropathologist in 86 percent of the 414 fatally injured road users in the sample, including 24 cases that were examined microscopically. There were no cases in which there was an injury to the brain in the absence of evidence of an impact to the head.},
keywords = {*Brain Injuries/et [Etiology], Acceleration, Accidents, autopsy, Brain Injuries/mo [Mortality], cause of death, Head, Humans, Mechanical, Nonpenetrating, Stress, Traffic, Wounds},
pubstate = {published},
tppubtype = {article}
}
Davis, A E
Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations Journal Article
In: Critical Care Nursing Quarterly, vol. 23, no. 3, pp. 1–13, 2000.
@article{Davis2000,
title = {Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations},
author = {Davis, A E},
year = {2000},
date = {2000-01-01},
journal = {Critical Care Nursing Quarterly},
volume = {23},
number = {3},
pages = {1--13},
abstract = {Traumatic brain injury (TBI) is a public health problem of great concern, because it affects more than 2 million individuals each year. TBI occurs as a result of motor vehicle crashes, falls, and sports-related events. Biomechanical mechanisms occurring at the time of the injury initiate primary and secondary injuries that evolve over several days. In this article the relationship between an blunt injury event and the subsequent damage produced is addressed. Mechanisms of brain injury from biomechanics to cellular pathobiology are presented. Primary and secondary injuries are differentiated, and specific focal and diffuse clinical syndromes are described. Cellular mechanisms responsible for injury are also addressed, because they provide the unifying concepts across the many clinical syndromes so often discussed separately in reviews of traumatic brain injury. [References: 26]},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
McLean, A J
Brain injury without head impact? Journal Article
In: Journal of Neurotrauma, vol. 12, no. 4, pp. 621–625, 1995.
@article{McLean1995,
title = {Brain injury without head impact?},
author = {McLean, A J},
year = {1995},
date = {1995-01-01},
journal = {Journal of Neurotrauma},
volume = {12},
number = {4},
pages = {621--625},
abstract = {The proposition that acceleration of the brain without direct impact to the head can result in brain injury is examined by reviewing a series of 414 road users who were fatally injured in the vicinity of Adelaide, South Australia. The series comprises 170 pedestrians, 10 pedal cyclists, 143 motorcyclists, and 91 vehicle occupants. In each case a member of the research team attended the autopsy to look for evidence of impact on the body, particularly to the head or face. The brain was examined by a neuropathologist and the type and pattern of injury was recorded. The circumstances of the crash were investigated, including an examination of the crash site and the vehicles involved and, where relevant, interviews with witnesses. In cases involving a motorcyclist the helmet worn was retrieved by the police and assigned to the research unit for examination. Particular attention was paid to the identification of objects causing injury to the head or face and also to objects impacted by a helmet. Brain injury was recorded as a cause of death in 55% of the 403 cases for which there was a clear classification of cause of death. Brain injury, at any level of severity, was identified by a neuropathologist in 86 percent of the 414 fatally injured road users in the sample, including 24 cases that were examined microscopically. There were no cases in which there was an injury to the brain in the absence of evidence of an impact to the head.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Davis, A E
Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations Journal Article
In: Critical Care Nursing Quarterly, vol. 23, no. 3, pp. 1–13, 2000.
Abstract | BibTeX | Tags: *Brain Injuries/pa [Pathology], *Brain Injuries/pp [Physiopathology], Biomechanical Phenomena, Brain Injuries/cl [Classification], Brain Injuries/co [Complications], Brain/me [Metabolism], Brain/pa [Pathology], Humans, intracranial hemorrhage, Neurons/me [Metabolism], Neurons/pa [Pathology], Nonpenetrating/pa [Pathology], Nonpenetrating/pp [Physiopathology], Skull Fractures/pa [Pathology], Skull Fractures/pp [Physiopathology], Traumatic/pa [Pathology], Traumatic/pp [Physiopatho, Wounds
@article{Davis2000,
title = {Mechanisms of traumatic brain injury: biomechanical, structural and cellular considerations},
author = {Davis, A E},
year = {2000},
date = {2000-01-01},
journal = {Critical Care Nursing Quarterly},
volume = {23},
number = {3},
pages = {1--13},
abstract = {Traumatic brain injury (TBI) is a public health problem of great concern, because it affects more than 2 million individuals each year. TBI occurs as a result of motor vehicle crashes, falls, and sports-related events. Biomechanical mechanisms occurring at the time of the injury initiate primary and secondary injuries that evolve over several days. In this article the relationship between an blunt injury event and the subsequent damage produced is addressed. Mechanisms of brain injury from biomechanics to cellular pathobiology are presented. Primary and secondary injuries are differentiated, and specific focal and diffuse clinical syndromes are described. Cellular mechanisms responsible for injury are also addressed, because they provide the unifying concepts across the many clinical syndromes so often discussed separately in reviews of traumatic brain injury. [References: 26]},
keywords = {*Brain Injuries/pa [Pathology], *Brain Injuries/pp [Physiopathology], Biomechanical Phenomena, Brain Injuries/cl [Classification], Brain Injuries/co [Complications], Brain/me [Metabolism], Brain/pa [Pathology], Humans, intracranial hemorrhage, Neurons/me [Metabolism], Neurons/pa [Pathology], Nonpenetrating/pa [Pathology], Nonpenetrating/pp [Physiopathology], Skull Fractures/pa [Pathology], Skull Fractures/pp [Physiopathology], Traumatic/pa [Pathology], Traumatic/pp [Physiopatho, Wounds},
pubstate = {published},
tppubtype = {article}
}
McLean, A J
Brain injury without head impact? Journal Article
In: Journal of Neurotrauma, vol. 12, no. 4, pp. 621–625, 1995.
Abstract | BibTeX | Tags: *Brain Injuries/et [Etiology], Acceleration, Accidents, autopsy, Brain Injuries/mo [Mortality], cause of death, Head, Humans, Mechanical, Nonpenetrating, Stress, Traffic, Wounds
@article{McLean1995,
title = {Brain injury without head impact?},
author = {McLean, A J},
year = {1995},
date = {1995-01-01},
journal = {Journal of Neurotrauma},
volume = {12},
number = {4},
pages = {621--625},
abstract = {The proposition that acceleration of the brain without direct impact to the head can result in brain injury is examined by reviewing a series of 414 road users who were fatally injured in the vicinity of Adelaide, South Australia. The series comprises 170 pedestrians, 10 pedal cyclists, 143 motorcyclists, and 91 vehicle occupants. In each case a member of the research team attended the autopsy to look for evidence of impact on the body, particularly to the head or face. The brain was examined by a neuropathologist and the type and pattern of injury was recorded. The circumstances of the crash were investigated, including an examination of the crash site and the vehicles involved and, where relevant, interviews with witnesses. In cases involving a motorcyclist the helmet worn was retrieved by the police and assigned to the research unit for examination. Particular attention was paid to the identification of objects causing injury to the head or face and also to objects impacted by a helmet. Brain injury was recorded as a cause of death in 55% of the 403 cases for which there was a clear classification of cause of death. Brain injury, at any level of severity, was identified by a neuropathologist in 86 percent of the 414 fatally injured road users in the sample, including 24 cases that were examined microscopically. There were no cases in which there was an injury to the brain in the absence of evidence of an impact to the head.},
keywords = {*Brain Injuries/et [Etiology], Acceleration, Accidents, autopsy, Brain Injuries/mo [Mortality], cause of death, Head, Humans, Mechanical, Nonpenetrating, Stress, Traffic, Wounds},
pubstate = {published},
tppubtype = {article}
}