Sorry, no publications matched your criteria.
Edlow, B L; Hinson, H E
Blowing the whistle on sports concussions Journal Article
In: Neurology, vol. 85, no. 17, pp. 1442–1443, 2015.
@article{Edlow2015,
title = {Blowing the whistle on sports concussions},
author = {Edlow, B L and Hinson, H E},
doi = {10.1212/WNL.0000000000001902},
year = {2015},
date = {2015-01-01},
journal = {Neurology},
volume = {85},
number = {17},
pages = {1442--1443},
abstract = {On March 13, 2015, Chris Borland, a star rookie linebacker on the San Francisco 49ers, announced his early retirement from professional football, citing concerns about chronic traumatic encephalopathy (CTE). Borland, who had a history of 2 diagnosed concussions, walked away from a multi-million-dollar contract and potential sports superstardom, explaining that "from what I've researched and what I've experienced, I don't think it's worth the risk." 1 Perhaps just as surprising as Borland's announcement was the support he received from teammates and other athletes-support that reflects a growing recognition in the athletic community that repetitive head trauma may be associated with CTE and other forms of dementia. 2 Indeed, in a recent legal settlement, the National Football League estimated that approximately 30% of its former players will develop dementia. At the high school and college levels, state legislatures and universities have enacted limits to the number of full-contact practices, citing similar concerns about concussions. These developments have prompted a societal conversation about the risks of contact sports. © 2015 American Academy of Neurology.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Levin, B; Bhardwaj, A
Chronic traumatic encephalopathy: A critical appraisal Journal Article
In: Neurocritical Care, vol. 20, no. 2, pp. 334–344, 2014.
@article{Levin2014,
title = {Chronic traumatic encephalopathy: A critical appraisal},
author = {Levin, B and Bhardwaj, A},
url = {http://www.scopus.com/inward/record.url?eid=2-s2.0-84896549537\&partnerID=40\&md5=138104db42f7ca99527a78bb9c821f59},
doi = {10.1007/s12028-013-9931-1},
year = {2014},
date = {2014-01-01},
journal = {Neurocritical Care},
volume = {20},
number = {2},
pages = {334--344},
abstract = {Chronic traumatic encephalopathy (CTE) formerly known as dementia pugilistica is a long-term neurodegenerative disorder associated with repeated subconcussive head injuries in high-contact sports. We reviewed the existing literature on CTE and examined epidemiological trends, risk factors, and its temporal progression, and proposed the underlying pathophysiological mechanisms that may provide unique insights to clinicians with an in-depth understanding of the disease to aid in the diagnosis and prevention, and provide future perspectives for research via search of Medline and Cochrane databases as well as manual review of bibliographies from selected articles and monographs. The prevalence of CTE in recent years is on the rise and almost exclusively affects men, with pathologic signs characterized by progressive memory loss, behavioral changes, and violent tendencies with some patients demonstrating Parkinsonian-like symptoms and signs. Many patients with CTE die following suicide, accident, or complications of drug or alcohol use. Postmortem pathologic analysis is characterized by neurofibrillary tangles and A$beta$ plaques in 50 % of cases. Currently, there are no ante-mortem diagnostic criteria, but modern imaging techniques such as functional magnetic resonance (MR) imaging, MR spectroscopy, and diffusion tension imaging hold promise for delineating the future diagnostic criteria. Further long-term longitudinal studies are warranted to investigate risk factors that will enhance understanding of the disease progression and its pathogenesis. © 2013 Springer Science+Business Media.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Andrikopoulos, J; Moines, D; Montenigro, P H; Stern, R A
Clinical presentation of chronic traumatic encephalopathy Journal Article
In: Neurology, vol. 83, no. 21, pp. 1991–1993, 2014.
@article{Andrikopoulos2014a,
title = {Clinical presentation of chronic traumatic encephalopathy},
author = {Andrikopoulos, J and Moines, D and Montenigro, P H and Stern, R A},
url = {http://www.scopus.com/inward/record.url?eid=2-s2.0-84922482119\&partnerID=40\&md5=57625e87083b405a16dda4c0a62ac42e},
year = {2014},
date = {2014-01-01},
journal = {Neurology},
volume = {83},
number = {21},
pages = {1991--1993},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Andrikopoulos, J
In: Journal of Neuropathology and Experimental Neurology, vol. 73, no. 4, pp. 375, 2014.
@article{Andrikopoulos2014,
title = {Correspondence regarding chronic traumatic encephalopathy in athletes: Progressive tauopathy following repetitive concussion. J Neuropathol Exp Neurol 2009;68: 709-35},
author = {Andrikopoulos, J},
url = {http://www.scopus.com/inward/record.url?eid=2-s2.0-84897451593\&partnerID=40\&md5=f463487f44a2ebf124b57a70320560a8},
doi = {10.1097/NEN.0000000000000057},
year = {2014},
date = {2014-01-01},
journal = {Journal of Neuropathology and Experimental Neurology},
volume = {73},
number = {4},
pages = {375},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Antonius, D; Mathew, N; Picano, J; Hinds, A; Cogswell, A; Olympia, J; Brooks, T; Di Giacomo, M; Baker, J; Willer, B; Leddy, J
In: Journal of Neuropsychiatry and Clinical Neurosciences, vol. 26, no. 4, pp. 313–322, 2014.
@article{Antonius2014,
title = {Behavioral health symptoms associated with chronic traumatic encephalopathy: A critical review of the literature and recommendations for treatment and research},
author = {Antonius, D and Mathew, N and Picano, J and Hinds, A and Cogswell, A and Olympia, J and Brooks, T and {Di Giacomo}, M and Baker, J and Willer, B and Leddy, J},
url = {http://www.scopus.com/inward/record.url?eid=2-s2.0-84920996232\&partnerID=40\&md5=cb8a1deab38101900f8d7a8ac0b7a80c},
year = {2014},
date = {2014-01-01},
journal = {Journal of Neuropsychiatry and Clinical Neurosciences},
volume = {26},
number = {4},
pages = {313--322},
abstract = {Chronic traumatic encephalopathy (CTE) is a neurodegenerative syndrome that has been linked to serious psychiatric symptoms, including depression, aggression, and suicidal behavior. This review critically examines the extant research on the behavioral manifestations of CTE and concludes that the paucity of longitudinal prospective studies on CTE, combined with a lack of research-accepted diagnostic criteria for identifying individuals who are considered at risk for CTE, makes it difficult to reliably establish a causal relationship between CTE and the onset of behavioral health problems. Selection and reporting bias and inconsistency in data collection methods are other concerns. To advance the field, there is a critical need for more empirical research on the behavioral manifestations of CTE. Recommendations and intervention models are also discussed. © 2014 American Psychiatric Association.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Stein, T D; Alvarez, V E; McKee, A C
Chronic traumatic encephalopathy: A spectrum of neuropathological changes following repetitive brain trauma in athletes and military personnel Journal Article
In: Alzheimer's Research and Therapy, vol. 6, no. 1, 2014.
@article{Stein2014,
title = {Chronic traumatic encephalopathy: A spectrum of neuropathological changes following repetitive brain trauma in athletes and military personnel},
author = {Stein, T D and Alvarez, V E and McKee, A C},
url = {http://www.scopus.com/inward/record.url?eid=2-s2.0-84892718392\&partnerID=40\&md5=c39a0e58ad33cee7a570b4681131d6ea},
doi = {10.1186/alzrt234},
year = {2014},
date = {2014-01-01},
journal = {Alzheimer's Research and Therapy},
volume = {6},
number = {1},
abstract = {Chronic traumatic encephalopathy (CTE) is a progressive neurodegenerative disease that occurs in association with repetitive traumatic brain injury experienced in sport and military service. In most instances, the clinical symptoms of the disease begin after a long period of latency ranging from several years to several decades. The initial symptoms are typically insidious, consisting of irritability, impulsivity, aggression, depression, short-term memory loss and heightened suicidality. The symptoms progress slowly over decades to include cognitive deficits and dementia. The pathology of CTE is characterized by the accumulation of phosphorylated tau protein in neurons and astrocytes in a pattern that is unique from other tauopathies, including Alzheimer's disease. The hyperphosphorylated tau abnormalities begin focally, as perivascular neurofibrillary tangles and neurites at the depths of the cerebral sulci, and then spread to involve superficial layers of adjacent cortex before becoming a widespread degeneration affecting medial temporal lobe structures, diencephalon and brainstem. Most instances of CTE (\>85% of cases) show abnormal accumulations of phosphorylated 43 kDa TAR DNA binding protein that are partially colocalized with phosphorylated tau protein. As CTE is characterized pathologically by frontal and temporal lobe atrophy, by abnormal deposits of phosphorylated tau and by 43 kDa TAR DNA binding protein and is associated clinically with behavioral and personality changes, as well as cognitive impairments, CTE is increasingly categorized as an acquired frontotemporal lobar degeneration. Currently, some of the greatest challenges are that CTE cannot be diagnosed during life and the incidence and prevalence of the disorder remain uncertain. Furthermore, the contribution of age, gender, genetics, stress, alcohol and substance abuse to the development of CTE remains to be determined. © 2014 BioMed Central Ltd.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Duhaime, A C
Response Journal Article
In: Journal of Neurosurgery, vol. 117, no. 6, pp. 1090–1091, 2012, ISBN: 0022-3085 1933-0693.
@article{Duhaime2012,
title = {Response},
author = {Duhaime, A C},
isbn = {0022-3085
1933-0693},
year = {2012},
date = {2012-01-01},
journal = {Journal of Neurosurgery},
volume = {117},
number = {6},
pages = {1090--1091},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Rabb, C H
Comments Journal Article
In: Neurosurgery, vol. 67, no. 4, pp. 1027–1028, 2010, ISBN: 0148-396X.
@article{Rabb2010,
title = {Comments},
author = {Rabb, C H},
isbn = {0148-396X},
year = {2010},
date = {2010-01-01},
journal = {Neurosurgery},
volume = {67},
number = {4},
pages = {1027--1028},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Sorry, no publications matched your criteria.